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The ERK pathway can also be activated by CAF-derived POSTN, leading to enhanced proliferation, migration, and EMT of NSCLC and gastric cancer cells [157, 158]

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2 , 4 Whereas UV‐associated radiation is a primary cause of DNA damage and melanoma, UV radiation‐induced MC1R activation can be protective against melanoma by enhancing DNA repair pathways (i.e

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